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Because GLP-2 acts through subepithelial myofibroblasts, which are the same cell population implicated in intestinal fibrogenesis, the risk-benefit profile of GLP-2 agonism may differ substantially between mucosal UC, inflammatory ileal CD, and stricturing or penetrating CD phenotypes, and this phenotypic stratification should be a required feature of future trial design

The first is conflating injectable and topical dosing conventions applying an injectable milligram-target approach to a topical protocol that should use concentration-based formulation, or vice versa

Images were analyzed using FIJI, descriptions of the number of bacteria counted are described in figure legends