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Cardiomyocyte survival promotion through ILK/PI3K/Akt anti-apoptotic signalling, cardiac progenitor cell activation and migration toward the ischaemic injury border zone, angiogenesis stimulation in the peri-infarct region to restore oxygen delivery to at-risk myocardium, and anti-fibrotic effects that attenuate the non-functional scar tissue formation that impairs cardiac function following myocardial infarction

10.1038/nrdp.2015.56 149 MelliosN.SugiharaH.CastroJ.BanerjeeA.LeC.KumarA.et al

10.1038/s41419-019-1734-7 258 YangQ.ZhaoQ.YinY

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