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Hepcidin prevents iron overload by reducing serum iron concentration through binding to the Ferroportin (FPN) on the basolateral aspect of the intestinal epithelium and the plasma membrane of macrophages (Nicolas et al., 2002)

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S-adenosylmethionine (SAMe) SAMe is an endogenous molecule formed from the amino acid L-methionine and ATP by the enzyme SAMe synthetase and initiates three metabolic pathways (Figure 1): 1

Mechanism of hyperammonemia in patients with GDH-HI In hepatocytes, elevated GDH activity leads to an increased rate of glutamate decomposition to ammonia