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Furthermore, the lack of cystathionine -synthase seemingly upregulates the expression of genes with regard to ER stress, hepatic lipid homeostasis, as well as genes linked to hepatic steatosis (57), whereas knockout of cystathionine -lyase results in decreased hepatic lipolysis (58)

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Conversely, stress conditions inhibit mTOR, triggering autophagy and metabolic reprogramming that sensitize cells to ferroptosis

Evidence for de novo morphine biosynthesis in animal cells, together with studies identifying dopamine as a key intermediate in that pathway, has supported the view that morphinergic processes are intrinsic to animal physiology rather than accidental reflections of dietary or plant exposure [1-5]