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Description

Molecular Mechanisms and Clinical Implications of Reversible Protein S -Glutathionylation

Reducing the global burden of cardiovascular disease, part 2: prevention and treatment of cardiovascular disease

Key mechanistic features: Selective GHSR-1a agonism Potent GH release without cortisol/ACTH/prolactin elevation Gq/11 Ca signalling Intracellular calcium rise triggers GH exocytosis Pulsatile GH release Preserves physiological GH pulse pattern