Mice heterozygous for mitochondrial trifunctional protein have compromised mitochondrial -oxidation and develop hepatic steatosis alongside a compensatory upregulation of CYP2E1-facilitated FAO and oxidative stress [101, 102]
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Proteobacteria, Actinobacteria, Fusobacteria and Verrucomicrobia make up the remaining 10% together with some species from the Archaea domain
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