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Alterations in NAD metabolism are critically linked to neurodegenerative disorders, as the accumulation of nicotinamide mononucleotide exerts pro-degenerative effects on axons, whereas enhancing its metabolism may rescue axons from degeneration (53)

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Results: Both compounds produced dose-dependent increases in mechanical withdrawal thresholds (antiallodynic effects), with ED 50 values of 2.45 0.23 mg/kg for pregabalin and 57.49 5.59 mg/kg for thioctic acid

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